“Vitamin K” is a term used to represent a group of chemically similar fat-soluble compounds that differ in their origin and function. In this regard, the main form of vitamin K, which is also considered the best source, is menaquinone 7 (vitamin K2).

The primary source of vitamin K2 is Natto, a popular Japanese food rich in enzymes that aid digestion (nattokinase). It is a compound produced only in the trans form through the natural fermentation of boiled soybeans by the action of a specific bacterium, Bacillus subtilis spp. natto, and contains approximately 1,000 μg of MK7 (menaquinone 7) per 100 g.

In its MK7 form, vitamin K2 is essential for blood clotting and plays an important role in calcium absorption and bone formation.

Vitamin K deficiency — especially a lack of vitamin K2 — can result in a phenomenon known as the “calcium paradox,” where very little calcium remains in weak bones, while excess of this mineral accumulates in the arteries, making them stiff and non-elastic.

In conjunction with vitamin D, Natomenna® (MK7) contributes to better calcium transport and absorption by the bones.

Vitamin K2 Nattomena® (MK7) is a natural source obtained through an innovative and patented fermentation production technology, resulting in a product that is more effective due to its high stability, concentration, and bioavailability, ensuring a prolonged effect.

Thanks to this production process, we offer the market Vitamin K2 Nattomena®, a fully trans(*) and GMO-Free product, available in various concentrations for use in a wide range of applications, being soluble in both water and oil.

All clinical trial data and published results were conducted with the fermented Vitamin K2, such as Nattomena®.

Additionally, Vitamin K2 Nattomena® holds the most important international certifications ensuring the quality of the product.
(*) The trans isomer of MK7 is primarily responsible for the biological activity of the vitamin and plays a crucial role in bone and cardiovascular health.

Cis analogs are considered biologically inactive.